A Way To Suppressing Prolactin

Sexual Reboot Forum A Way To Suppressing Prolactin

This topic contains 9 replies, has 1 voice, and was last updated by  Jeff 7 years ago.

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    Effect of pyridoxine on human hypophyseal trophic hormone release: a possible stimulation of hypothalamic dopaminergic pathway.

    A single dose of pyridoxine (300 mg iv) produced significant rises in peak levels of immunoreactive growth hormone GH and significant decrease of plasma prolactin PRL in 8 hospitalized healthy subjects. Serum glucose, luteinizing hormone LH, follicle stimulating hormone FSH and thyrotropin TSH were not altered significantly. In addition, in 5 acromegalic patients who were studied with both L-dopa and pyridoxine, inhibition of GH secretion followed either agent in a similar pattern. These data suggest a hypothalamic dopaminergic effect of pyridoxine.

    Pyridoxine (B6) suppresses the rise in prolactin and increases the rise in growth hormone induced by exercise.

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    Hi all, please always put the original link when putting info like this on the forum. It makes research much easier!



    yea man good point

    sorry about that people-cant find link now either

    hope that infomation aids those of you who plan to do further reseach into this matter.



    Well I don’t know about that drug but I have given up on Vitex for prolactin suppression and I still have quite a few bottles. I’m not using them, perhaps I should donate to other members who might want to try it? Only thing is the shipping.

    As far as prolactin suppressing, I’ll go with boosting dopamine and testosterone for now. That will definitely decrease prolactin.



    ive been getting a 100mg dose of b6 a day through other suppliments-its mixed with them

    not noticed anything tbh-much higher dose needed-can give you tingle and numb sensations in the hands and feet which is somthing that concerns me.

    it will be some time before im stable enough to stimulate dopamine production-so im looking for ways to combat this problem before hand.



    I also want to supress prolactin, if possible 100%. After searching ages on the internet, their is very little information on how to inhibit or supress prolactin.

    But typing in “Hyperprolactinemia” the medical term for excessive prolactin in the bloodstream, I found some possible solutions; only problem being is its regulated medicine, but good info non the less.


    # Medication


    * The dopamine agonist, bromocriptine mesylate, is the initial drug of choice. It lowers the prolactin level in 70-100% of patients. Agents other than bromocriptine have been used (eg, cabergoline, quinagolide). Cabergoline and pergolide are available in the United States. Cabergoline, in particular, probably is more effective and causes fewer adverse effects than bromocriptine. However, it is much more expensive. Cabergoline often is used in patients who cannot tolerate the adverse effects of bromocriptine or in those who do not respond to bromocriptine. Pergolide has not been approved by the US Food and Drug Administration (FDA) for use in patients with this condition.

    * Response to therapy should be monitored by checking fasting serum prolactin levels and checking tumor size with MRI. Most women (approximately 90%) regain cyclic menstruation and achieve resolution of galactorrhea. Testosterone levels in men increase but may remain below normal.

    * Therapy should be continued for approximately 12-24 months (depending on the degree of symptoms or tumor size) and then withdrawn if prolactin levels have returned to the normal range. After withdrawal, approximately one sixth of patients maintain normal prolactin levels.

    * Bromocriptine also is used to shrink macroadenomas. Normalization of visual fields is observed in as many as 90% of patients. A failure to improve within 1-3 months is an indication for surgery. Tumors usually shrink to 50% of their original size in approximately 90% of patients treated for macroadenomas for 1 year. In patients with nonprolactinoma tumors (masses that are compressing the pituitary stalk), medical treatment reduces serum prolactin levels but does not reduce tumor size. Cabergoline is somewhat more effective than bromocriptine in terms of tumor shrinkage.



    Basically we need dopamine agonists.


    My guess high levels of l-dopa would be the solution.



    Found some more info.


    “An herbal supplement called mucuna pruriens elevates dopamine levels and it has been used in India for many centuries for Parkinson’s disease.”

    I think increasing dopamine info will be easy to find, its just increasing it a lot at once is what I would like to achieve, like the medicine they give parkinson disease patients.



    I believe lack of dopamine is the reason for my low libido and I’m seriously considering trying dopamine agonists.

    On Thundersplace.com there’s interesting thread about Cabergoline. People there have experimented with small doses of it and report mixed results. They have ordered the drug from an UK based firm:


    If taken at 0.5mg doses every third day the little bottle will last for a year. I wonder if 0.5mg would suffice in the long run or if the dose needs to be increased for it to be effective.



    We also need to look into the safety of these prescription dopamine medications. Do they affect natural dopamine production? Are there any side effects? etc.

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